- First-degree burn.
- Second-degree burn (partial thickness).
- Third-degree burn (full thickness).
- Fourth-degree burn.
No category found.
- Pitting edema.
- Non-pitting edema.
- Lymphedema.
- Dependent edema.
- Increased stomach acid.
- Impaired production of pancreatic enzymes for digestion.
- Excessive bile production.
- Rapid intestinal motility.
- Autosomal recessive.
- Autosomal dominant.
- X-linked recessive.
- X-linked dominant.
- Decreased blood pressure.
- Increased blood pressure and fluid retention.
- Vasodilation.
- Decreased sodium reabsorption.
- Hypertrophy.
- Hyperplasia.
- Metaplasia.
- Atrophy.
- Chronic and persistent.
- Sudden onset, short duration, and localized.
- Difficult to treat with analgesics.
- Not associated with tissue damage.
- Curable.
- Acute and rapidly progressive.
- Chronic with periods of increased and decreased activity.
- Always fatal.
- Mature neutrophils.
- Immature neutrophils (bands).
- Lymphocytes.
- Eosinophils.
- Reversible change of one cell type to another.
- Cells increasing in size due to increased workload.
- Disorganized cell growth, varying in size, shape, and organization.
- A normal, adaptive response.
- Von Willebrand disease.
- Disseminated intravascular coagulation (DIC).
- Hemophilia A.
- Hemophilia B.
- Prostaglandins.
- Histamine and leukotrienes.
- Complement proteins.
- Antibodies.
- Benign tumor.
- Malignant tumor.
- Hypertrophic lesion.
- Metaplastic change.
- Respiratory acidosis.
- Metabolic acidosis.
- Respiratory alkalosis.
- Metabolic alkalosis.
- Decreased blood flow to the lungs.
- Increased pressure in the pulmonary arteries from left heart failure.
- Bronchospasm.
- Alveolar destruction.
- Stage I.
- Stage II.
- Stage III.
- Stage IV.
- Cells decrease in size.
- Cells increase in number.
- One mature cell type is replaced by another mature cell type.
- Cells become disorganized and abnormal.
- Coagulative necrosis.
- Liquefactive necrosis.
- Caseous necrosis.
- Fat necrosis.
- Insulin.
- Cortisol.
- Growth hormone.
- Estrogen.
