- Excessive clot formation leading to widespread microvascular thrombosis and consumption of clotting factors, followed by bleeding.
- Only excessive bleeding with no clot formation.
- Increased production of platelets.
- Decreased fibrinolysis.
No category found.
- Autosomal dominant.
- Autosomal recessive.
- X-linked dominant.
- X-linked recessive.
- Increased intravascular protein synthesis.
- Decreased capillary permeability.
- Increased capillary hydrostatic pressure and increased capillary permeability.
- Decreased lymphatic drainage.
- Bronchial smooth muscle relaxation and decreased mucus production.
- Bronchospasm, airway inflammation, and increased mucus production.
- Destruction of alveolar walls.
- Fluid accumulation in the pleural space.
- Rupture of a cerebral blood vessel.
- An embolus or thrombus obstructing cerebral blood flow.
- Vasospasm of cerebral arteries.
- Hypoxia due to respiratory failure.
- Alpha cells.
- Beta cells.
- Delta cells.
- Acinar cells.
- Respiratory acidosis.
- Metabolic acidosis.
- Respiratory alkalosis.
- Metabolic alkalosis.
- The rapid proliferation of genetically altered cells.
- The irreversible genetic alteration of a cell by a carcinogen.
- The spread of cancer cells to distant sites.
- The formation of a visible tumor.
- Malignant tumor.
- Benign tumor.
- Carcinoma in situ.
- Sarcoma.
- Increased urine output and hypernatremia.
- Decreased urine output and hyponatremia.
- Normal fluid balance with hyperkalemia.
- Fluid volume deficit and hypercalcemia.
- Fatty infiltration with normal liver architecture.
- Widespread fibrosis and nodule formation, disrupting liver structure and function.
- Acute inflammation with viral replication.
- Increased bile production.
- Direct bacterial infection.
- Friction and shear forces causing tissue deformation and ischemia.
- Allergic reaction to bedding material.
- Excessive moisture leading to maceration.
- Massive vasodilation and increased capillary permeability due to inflammatory mediators.
- Direct myocardial tissue damage.
- Pulmonary embolism.
- Hyperglycemia due to insulin resistance.
- Increased red blood cell destruction.
- Decreased iron absorption in the gut.
- Impaired production of erythropoietin by the kidneys.
- Excessive blood loss through dialysis.
- Decreased oxygen demand by the heart.
- Inadequate oxygen supply to the myocardium.
- Increased venous return to the heart.
- Pulmonary hypertension.
- Decrease blood flow to the injured area.
- Reduce capillary permeability.
- Increase blood flow to the injured area, facilitating delivery of immune cells.
- Constrict arterioles to limit swelling.
- Excessive insulin production leading to hypoglycemia.
- Absolute or relative insulin deficiency leading to increased fat metabolism and ketone body formation.
- Excessive fluid intake leading to hyponatremia.
- Increased renal excretion of glucose without ketone production.
- Atrophy.
- Hyperplasia.
- Metaplasia.
- Hypertrophy.
- IgG antibodies.
- T-cytotoxic cells.
- IgE antibodies and mast cell degranulation.
- Immune complexes.
