- Curable.
- Acute and rapidly progressive.
- Chronic with periods of increased and decreased activity.
- Always fatal.
No category found.
- Mature neutrophils.
- Immature neutrophils (bands).
- Lymphocytes.
- Eosinophils.
- Reversible change of one cell type to another.
- Cells increasing in size due to increased workload.
- Disorganized cell growth, varying in size, shape, and organization.
- A normal, adaptive response.
- Von Willebrand disease.
- Disseminated intravascular coagulation (DIC).
- Hemophilia A.
- Hemophilia B.
- Prostaglandins.
- Histamine and leukotrienes.
- Complement proteins.
- Antibodies.
- Benign tumor.
- Malignant tumor.
- Hypertrophic lesion.
- Metaplastic change.
- Respiratory acidosis.
- Metabolic acidosis.
- Respiratory alkalosis.
- Metabolic alkalosis.
- Decreased blood flow to the lungs.
- Increased pressure in the pulmonary arteries from left heart failure.
- Bronchospasm.
- Alveolar destruction.
- Stage I.
- Stage II.
- Stage III.
- Stage IV.
- Cells decrease in size.
- Cells increase in number.
- One mature cell type is replaced by another mature cell type.
- Cells become disorganized and abnormal.
- Coagulative necrosis.
- Liquefactive necrosis.
- Caseous necrosis.
- Fat necrosis.
- Insulin.
- Cortisol.
- Growth hormone.
- Estrogen.
- Bone pain and kidney stones.
- Muscle weakness and lethargy.
- Tetany, muscle spasms, and positive Chvostek's sign.
- Constipation.
- Father to son.
- Mother to daughter.
- Affected mother to all children.
- Carrier mother to affected son.
- Localized inflammation.
- Systemic manifestation of inflammation.
- Cellular adaptation.
- Genetic defect.
- Acute hyperglycemia.
- Damage to small blood vessels by chronic hyperglycemia.
- Hypoglycemia.
- Protein deficiency.
- Fibroblasts only.
- Myofibroblasts.
- Macrophages.
- Epithelial cells.
- Neutrophils.
- Macrophages and lymphocytes.
- Eosinophils.
- Basophils.
- Decreased oxygen demand.
- Pulmonary congestion and impaired gas exchange.
- Bronchospasm.
- Metabolic acidosis.
