- Enhanced immune function.
- Decreased blood glucose levels.
- Impaired immune function and increased risk of infection.
- Improved sleep patterns.
Category: Pathophysiology
- Autoantibodies attacking the neuromuscular junction.
- Systemic inflammation mediated by immune complexes and T-cells, primarily affecting synovial joints.
- Destruction of pancreatic beta cells.
- Increased red blood cell destruction.
- Normal scar formation.
- Excessive collagen formation, extending beyond the original wound boundaries.
- Incomplete wound closure.
- Decreased tensile strength.
- White blood cells.
- Hemoglobin synthesis.
- Platelet production.
- Plasma volume.
- Increased capillary hydrostatic pressure.
- Decreased plasma oncotic pressure.
- Increased lymphatic drainage.
- Increased arterial blood flow.
- Bronchospasm.
- Massive pulmonary edema and severe hypoxemia due to capillary-alveolar membrane damage.
- Chronic airway inflammation.
- Alveolar wall destruction.
- Alveolar wall destruction.
- Airway inflammation, mucus gland hyperplasia, and excessive mucus production.
- Reversible bronchospasm.
- Decreased peripheral airway resistance.
- Primary intention.
- Secondary intention.
- Tertiary intention.
- First intention.
- Necrosis.
- Apoptosis.
- Autophagy.
- Metaplasia.
- The heart pumps blood efficiently.
- The heart's pumping ability is decreased.
- The heart valves are narrowed.
- The patient is experiencing fluid volume deficit.
- Direct damage to kidney tubules.
- Obstruction of urine outflow.
- Decreased blood flow to the kidneys.
- Glomerular inflammation.
- Red blood cells.
- Platelets.
- White blood cells.
- Plasma proteins.
- Gallstone obstruction only.
- Autodigestion of pancreatic tissue by its own enzymes.
- Increased insulin secretion.
- Liver inflammation.
- Klinefelter syndrome.
- Turner syndrome.
- Down syndrome.
- Cystic fibrosis.
- Atrophy.
- Hyperplasia.
- Metaplasia.
- Dysplasia.
- Initial sympathetic nervous system activation.
- Mobilization of resources and adaptation.
- Depletion of adaptation energy and potential for organ damage.
- Increased resistance to disease.
- Hypertension, obesity, and hyperlipidemia.
- Venous stasis, endothelial injury, and hypercoagulability.
- Hypovolemia, infection, and low blood pressure.
- Anemia, fever, and leukopenia.
- Continuous inflammation limited to the colon.
- Patchy, transmural inflammation that can affect any part of the GI tract from mouth to anus.
- Ulceration limited to the mucosal layer of the rectum.
- Stricture formation only in the esophagus.
- Undergo apoptosis.
- Shrink in size.
- Spread from the primary site to distant organs.
- Become well-differentiated.
